By R. Caplan Louis, Warren J. Manning
The single resource to completely hide each element of mind embolism, this advisor analyzes the motives, indicators, analysis, and administration of this disorder-providing a close evaluate of significant subject matters pertinent to embolism together with donor assets, recipient websites, embolic fabric, recipient mind provide arteries, vascular and mind pathology, and the remedy of cardiac and arterial assets. basically guiding readers via key anatomical buildings, diagnostic advancements, healing advances, and top practices in medical care, this resource will function a must have table reference for any clinician within the box.
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Extra info for Brain Embolism
45. Foix C, Hillemand P, Schalit I. Sur le syndrome lateral du bulbe et irrigation du bulbe superieur. Rev Neurol 1925; 41:160 – 179. 46. Foix C, Hillemand P, Ley J. Relativement ramollissement cerebral a sa frequence et a son siege et a l’importance relative des obliterations arterielles, completes ou incompletes dans sa pathogenie. Rev Neurol 1927; 43:217 – 218. 47. Kubik CS, Adams RD. Occlusion of the basilar artery—a clinical and pathological study. Brain 1946; 69:73 – 121. 48. Caplan LR. Posterior Circulation Disease.
145. McHenry L. Introduction to Willis T. The Anatomy of the Brain and Nerves. Birmingham: The Classics of Neurology and Neurosurgery Library, 1965. 146. Haymaker W, ed. The Founders of Neurology. Springfield, Ilinois: Charles C Thomas, 1953. 147. Roland CG. Introduction to Osler W. The Cerebral Palsies of Children and On Chorea and Choreiform Affections. Birmingham: The Classics of Neurology and Neurosurgery Library, 1986. 2 Overview: The Major Components of Brain Embolism Louis R. A. There are three important actors, dramatis personae, in the drama of brain embolism: the recipient artery that catches and receives the embolic material, even temporarily; the embolic material itself—the stuff that makes up the emboli; and the donor source from which the embolic material originates (Fig.
Imaging of patients with coronary artery disease showed that many had ventricular aneurysms or hypokinetic, or akinetic, segments that harbored thrombi (115,116). Echocardiography also showed that atrial septal dysfunction in the form of atrial septal defects, patent foramen ovale, and atrial septal aneurysms were more frequent than previously thought and that these lesions were very common in patients with strokes of uncertain cause (117,118). 18 Caplan Although it was widely known that the aorta was very often the seat of severe atherosclerosis, the role of aortic plaques as donor sources of brain embolism was only recently emphasized.